The H19/let-7 double-negative feedback loop contributes to glucose metabolism in muscle cells

نویسندگان

  • Yuan Gao
  • Fuju Wu
  • Jichun Zhou
  • Lei Yan
  • Michael J. Jurczak
  • Hui-Young Lee
  • Lihua Yang
  • Martin Mueller
  • Xiao-Bo Zhou
  • Luisa Dandolo
  • Julia Szendroedi
  • Michael Roden
  • Clare Flannery
  • Hugh Taylor
  • Gordon G. Carmichael
  • Gerald I. Shulman
  • Yingqun Huang
چکیده

The H19 lncRNA has been implicated in development and growth control and is associated with human genetic disorders and cancer. Acting as a molecular sponge, H19 inhibits microRNA (miRNA) let-7. Here we report that H19 is significantly decreased in muscle of human subjects with type-2 diabetes and insulin resistant rodents. This decrease leads to increased bioavailability of let-7, causing diminished expression of let-7 targets, which is recapitulated in vitro where H19 depletion results in impaired insulin signaling and decreased glucose uptake. Furthermore, acute hyperinsulinemia downregulates H19, a phenomenon that occurs through PI3K/AKT-dependent phosphorylation of the miRNA processing factor KSRP, which promotes biogenesis of let-7 and its mediated H19 destabilization. Our results reveal a previously undescribed double-negative feedback loop between sponge lncRNA and target miRNA that contributes to glucose regulation in muscle cells.

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عنوان ژورنال:

دوره 42  شماره 

صفحات  -

تاریخ انتشار 2014